Difference between revisions of "Team:CKWA-China/Human Practices"

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In addition to its role in tumor formation, MYC can also work with Sox2, Oct4 and KLF4 to reprogram fibroblasts to a pluripotent stem cell state. Based on its central role in cell growth, proliferation, tumor formation and stem cells, author Chi V. Dang states in the article that he takes these key questions such as what is the molecular function of Myc, the protein product of MYC, how does MYC contribute to tumor formation, the differences between the MYC proto-oncogene and the runaway forms found in various human cancers, and whether MYC or Myc's Can target genes be targets for cancer therapy?
 
In addition to its role in tumor formation, MYC can also work with Sox2, Oct4 and KLF4 to reprogram fibroblasts to a pluripotent stem cell state. Based on its central role in cell growth, proliferation, tumor formation and stem cells, author Chi V. Dang states in the article that he takes these key questions such as what is the molecular function of Myc, the protein product of MYC, how does MYC contribute to tumor formation, the differences between the MYC proto-oncogene and the runaway forms found in various human cancers, and whether MYC or Myc's Can target genes be targets for cancer therapy?
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Summary: c-Myc and Cancer
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<h3 class="point" id="point-2">Summary: c-Myc and Cancer</h3>
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Dysregulation of the proto-oncogene MYC has a key role in human tumorigenesis. Unlike other proto-oncogenes that are active due to mutation or truncation, MYC is dysregulated due to loss of transcriptional control, resulting in protein overexpression.
 
Dysregulation of the proto-oncogene MYC has a key role in human tumorigenesis. Unlike other proto-oncogenes that are active due to mutation or truncation, MYC is dysregulated due to loss of transcriptional control, resulting in protein overexpression.
 
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Revision as of 02:14, 20 October 2021

Human Practices